Anatomy: Acute Myeloid Leukemia Essay

Bence Toth Ms - Anatomy: Acute Myeloid Leukemia Essay introduction. Schmaeman Anatomy paper 2 10/25/12 Acut Myeloid Leukemia Acute myeloid leukemia is one of the most common type of blood cancer. This kind of leukemia usually develops from cells that would turn into white blood cells. Sometimes, though, it can develop from other types of blood-forming cells. Acute myeloid leukemia starts in the bone marrow. This is the soft inner parts of bones. With acute types of leukemia such as AML, bone marrow cells don’t mature the way they’re supposed to. These immature cells, usually called blast cells, and just keep building up. (webmd). The large body of bone marrow is inside the bone.

The marrow formed in the red blood cells, platelets and white blood cells of five figures. The most imature cells called blasts. The blasts are normally only a very few reach the bloodstream, the maturation process undergo bone marrow and in the circulation will have the mature cell forms. The white blood cells can be divided into two main groups: myelocyta and lymphocytes. The myelocytas also called granulocytes in the citoplasm granules (granules) basis. These include the neutrophil, basophil and eosinophil granulocytes and monocytes. These cells are responsible for the destruction of bacteria.

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The lymphocytes in the blood stream and lymphatic system are present. These control the infections occurring immune response and produce antibodies. (nursingdepartment) AML white blood cells in the bone marrow malignancies. Are a result of the genes (DNA) level is called. Translocation of the 9-named ABL gene on chromosome 22 with gene is located on chromosome repositioned. The resulting fusion gene is generated by the so-called protein. BCR-ABL tyrosine kinas fusion with a continuous signal to the abnormal proliferation of white blood cells result in the development of CML which is the natural course of disease end.

A three phases can be distinguished. Diagnosis of patients in the so-called 95%. chronic phase, which without treatment, usually 3-5 years. This is followed by the so-called accelerated phase (6-12 months), during which the disease progresses more quickly. The appearance of the accelerated phase may indicate deterioration of blood counts and worsening of symptoms (loss of appetite, bruising, bleeding onset). The final called. blastic phase (or blast crisis) continues to rise in the blood and bone marrow cancer of white blood cells (cancer cells), the number of serious blood formation is impaired.

The average survival of patients at this stage between 3-6 months. (netambulancia) This type of leukemia can spread quickly to the blood and to other parts of the body such as the brain and spinal cord, liver, spleen, lymph nodes, testicles. Males and smokers, especially smokers over age 60, have an increased risk of AML. Other risk factors include past chemotherapy or radiation therapy treatment. We can see in the research around 80 to 90 percent of children with acute myeloid leukemia attain remissions (absence of leukemic cells), some of those patients have later recurrences.

About 70 percent of children with AML achieve long-term remissions with chemotherapy or stem cell transplantation. The symptoms of AML are caused by replacement of normal bone marrow with leukemic cells, resulting in a drop in red blood cells, platelets, and normal white blood cells. These symptoms include fatigue, shortness of breath, easy bruising and bleeding, and increased risk of infection. The goal of treatment is to stop abnormal cell proliferation, especially in compiled according to establish protocol chemotherapy treatment is applied, which consists in the inhibition of cell division.

This case is still in the bone marrow transplant, which is carried out to allow the high-dose chemotherapy, toxic application will facilitate the addition of a healthy bone marrow hematopoietic recovery. The problem of rejection caused by bone marrow transplantation, which occurs depending on to what extent the transplanted bone marrow cells differ from the cells of the patient. It is therefore advisable to get parent or brother, because if the bone structure is very different, then the patient is recognized as foreign and destroy it. Careful hygiene, bacteria-free environment is a prerequisite; therefore, patients should be hospitalized during treatment. If necessary, platelet and red blood cell replacement must be provided. (egeszsegvilag)

Citation Akut Myeloid Leukemia(AML). N. p. , n. d. Web. 01 Nov. 2012. <http://www. egeszsegvilag. hu/betegseg/akut-myeloid-leukemia-aml>. Acute Myeloid Leukemia: Symptoms, Survival, Treatments, and More. ” WebMD. WebMD, n. d. Web. 01 Nov. 2012. <http://www. webmd. com/cancer/acute-myeloid-leukemia-symptoms-treatments>. The Nursing Department.  Pathophysiology of Acute Myeloid Leukemia ~. N. p. , n. d. Web. 01 Nov. 2012. <http://nursingdepartment. blogspot. com/2009/03/pathophysiology-of-acute-myeloid. html>. What Is the Patho Physiology of Acute Myeloid Leukemia? ” What Is the Patho Physiology of Acute Myeloid Leukemia? N. p. , n. d. Web. 01 Nov. 2012. <http://www. alldiseases. org/what-is-the-patho-physiology-of-acute-myeloid-leukemia/>. <http://www. netambulancia. hu/daganatos megbetegedesek/kronikus myeloid leukemia _cml_ kronikus granulocytas leukemia _cgl>.

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